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| 9. |  | FIERRO, S.; OLIVERA-MUZANTEA, J.; GIL, J.; VIÑOLES, C. Effects of prostaglandin administration on ovarian follicular dynamics, conception, prolificacy, and fecundity in sheep. Theriogenology, 2011, v.76, no.4, p.630-639. Article history: Received 23 August 2010 / Received in revised form 17 March 201 / Accepted 18 March 2011.| Biblioteca(s): INIA La Estanzuela; INIA Las Brujas. |
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| 11. | | MINTEGUIAGA, M. A.; LÓPEZ MAZZ, C.; FIERRO, S.; BANCHERO, G. Including magnesium sulfate in the diet of twin-bearing ewes in the last week of gestation improves the adaptation of lambs to extrauterine life. Animal Production Science, 2024, Volume 64, Issue 12, AN24022. https://doi.org/10.1071/AN24022 Article history: Received 2 February 2024; Accepted 14 July 2024; Published 2 August 2024. -- Correspondence: Minteguiaga, M.A.; Facultad de Veterinaria, UDELAR, Ruta 3 km 363, Paysandú, Uruguay; email:maurominteguiaga@gmail.com --...| Biblioteca(s): INIA Las Brujas. |
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| 15. | | PÉREZ, I.; AGUERRE, J. I.; CIAPPESONI, G.; FIERRO, S.; RAMOS, Z. Estrategia de control de los nematodos gastrointestinales en ovinos mediante sistemas de pastoreo. [Strategy for controlling gastrointestinal nematodes in sheep through grazing systems]. Sección: Ovinos, Caprinos y Camélidos Sudamericanos. PANVET 2024, XXVII Congreso Panamericano de Ciencias Veterinarias, "Una salud: Una voz desde todas las áreas de la profesión", 3-6 diciembre 2024, Montevideo (UY). Libro de Resúmenes. p.395-397.| Biblioteca(s): INIA Las Brujas. |
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| 16. | | MINTEGUIAGA, M.A.; BANCHERO, G.; FIERRO, S.; ADRIEN, M.L.; OLIVERA-MUZANTE, J. Impact of focus feeding on reproductive losses, prolificacy, or fecundity of estrous synchronized ewes. Livestock Science, 2022, Volume 256, Article number 104817. doi: https://doi.org/10.1016/j.livsci.2021.104817 Article history:Received 4 June 2021/ Revised 24 December 2021/ Accepted 27 December 2021/ Available online 30 December 2021.| Biblioteca(s): INIA La Estanzuela. |
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| 17. | | OLIVERA, J.; GIL, J.; FIERRO, S.; DURÁN, G.; ALABART, J.L. Pérdidas embrionarias entre el no retorno al servicio y la fertilidad a ecografía en ovinos bajo diferentes tecnologías de IA. ln: INIA Tacuarembó. Estación Experimental Glencoe. Proyecto Merino Fino del Uruguay: octava distribución de carneros generados en el núcleo fundacional de Merino fino de la Unidad Experimental Glencoe, 1999-2007, Paysandú, 13 de diciembre, 2006. Tacuarembó (Uruguay): INIA, 2007. p. 54-59 (INIA Serie Actividades de Difusión ; 523)| Biblioteca(s): INIA Tacuarembó. |
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| 18. | | GIL, J.; OLIVERA, J.; FIERRO, S.; DURÁN, G.; GAMARRA, J.; TEIXEIRA, V.; ARAÚJO, A.; STOLETNIY, G. Inseminación intrauterina con semen congelado en majadas Merino Fino: comparación de protocolos de sincronización estral. In: INIA Tacuarembó. Unidad Experimental Glencoe. Avances obtenidos en el Proyecto Merino Fino del Uruguay: núcleo fundacional Unidad Experimental Glencoe, 1999-2006. Paysandú, 19 diciembre, 2006. Tacuarembó (Uruguay): INIA, 2006. p. 16-20 (INIA Serie Actividades de Difusión ; 475)| Biblioteca(s): INIA Tacuarembó. |
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| 19. | | VIÑOLES, C.; DE BARBIERI, I.; GIL, J.; OLIVERA, J.; FIERRO, S.; BIALADE, F.; MONTOSSI, F. Long-term effect of nutrition on the metabolic status and reproductive potential of Merino rams under grazing conditions. Animal Production Science, 2012, v. 52, no.10, p. 881-889. https://doi.org/10.1071/AN11365 History article: Received 2 November 2011; accepted 17 March 2012; published online 16 July 2012.Corresponding author. Acknowledgements: The authors wish to thank the staff of INIA’s Research Station and the studentsin thesis from the...| Biblioteca(s): INIA Tacuarembó. |
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| 20. | | PIAGGIO, L; DESCHENAUX, H.; BALDI, F.; FIERRO, S.; QUINTANS, G.; BANCHERO, G. Plane of nutrition of Corriedale ewe lambs from foetal life to the onset of breeding affects weight at service and reproductive outcome. Animal Production Science, v. 55, n. 8, p. 1011-1017, 2015. Article history: Received 21 June 2013, accepted 27 May 2014, published online 15 September 2014.| Biblioteca(s): INIA La Estanzuela; INIA Treinta y Tres. |
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Registro completo
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Biblioteca (s) : |
INIA La Estanzuela. |
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Fecha actual : |
02/03/2017 |
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Actualizado : |
07/03/2017 |
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Autor : |
GARCIA, J.P; GIANNITTI, F.; FINNIE, J.W.; MANAVIS, J. .; BEINGESSER, J.; ADAMS ,V.; ROOD, J.I.; UZAL, F.A. |
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Afiliación : |
FEDERICO GIANNITTI, INIA (Instituto Nacional de Investigación Agropecuaria), Uruguay. |
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Título : |
Comparative Neuropathology of Ovine Enterotoxemia Produced by Clostridium perfringens Type D Wild-Type Strain CN1020 and Its Genetically Modified Derivatives. |
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Fecha de publicación : |
2015 |
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Fuente / Imprenta : |
Veterinary Pathology, 2015, v.52.n.6. p.1250-1253, 2015. |
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Idioma : |
Inglés |
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Contenido : |
Abstract
Clostridium perfringens type D causes enterotoxemia in sheep and goats. The disease is mediated by epsilon toxin (ETX), which affects the cerebrovascular endothelium, increasing vascular permeability and leading to cerebral edema. In the present study, we compared the distribution and severity of the cerebrovascular changes induced in lambs by C. perfringens type D strain CN1020, its isogenic etx null mutant, and the ETX-producing complemented mutant. We also applied histochemical and immunohistochemical markers to further characterize the brain lesions induced by ETX. Both ETX-producing strains induced extensive cerebrovascular damage that did not differ significantly between each other in nature, neuroanatomic distribution, or severity. By contrast, lambs inoculated with the etx mutant or sterile, nontoxic culture medium did not develop detectable brain lesions, confirming that the neuropathologic effects observed in these infections are dependent on ETX production. Lambs treated with the wild-type and complemented strains showed perivascular and mural vascular edema, as well as serum albumin extravasation, particularly severe in the cerebral white matter, midbrain, medulla oblongata, and cerebellum. Brains of animals inoculated with the ETX-producing strains showed decreased expression of glial fibrillary acidic protein and increased expression of aquaporin-4 in the end-feet processes of the astrocytes around blood vessels. Early axonal injury was demonstrated with anti?amyloid precursor protein immunohistochemistry. Perivascular accumulation of macrophages/microglia with intracytoplasmic albumin globules was also observed in these animals. This study demonstrates that ETX is responsible for the major cerebrovascular changes in C. perfringens type D?induced disease. MenosAbstract
Clostridium perfringens type D causes enterotoxemia in sheep and goats. The disease is mediated by epsilon toxin (ETX), which affects the cerebrovascular endothelium, increasing vascular permeability and leading to cerebral edema. In the present study, we compared the distribution and severity of the cerebrovascular changes induced in lambs by C. perfringens type D strain CN1020, its isogenic etx null mutant, and the ETX-producing complemented mutant. We also applied histochemical and immunohistochemical markers to further characterize the brain lesions induced by ETX. Both ETX-producing strains induced extensive cerebrovascular damage that did not differ significantly between each other in nature, neuroanatomic distribution, or severity. By contrast, lambs inoculated with the etx mutant or sterile, nontoxic culture medium did not develop detectable brain lesions, confirming that the neuropathologic effects observed in these infections are dependent on ETX production. Lambs treated with the wild-type and complemented strains showed perivascular and mural vascular edema, as well as serum albumin extravasation, particularly severe in the cerebral white matter, midbrain, medulla oblongata, and cerebellum. Brains of animals inoculated with the ETX-producing strains showed decreased expression of glial fibrillary acidic protein and increased expression of aquaporin-4 in the end-feet processes of the astrocytes around blood vessels. Early axonal injury was demonstrated wi... Presentar Todo |
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Thesagro : |
ENFERMEDADES DE LOS ANIMALES. |
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Asunto categoría : |
-- |
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Marc : |
LEADER 02512naa a2200217 a 4500
001 1056750
005 2017-03-07
008 2015 bl uuuu u00u1 u #d
100 1 $aGARCIA, J.P
245 $aComparative Neuropathology of Ovine Enterotoxemia Produced by Clostridium perfringens Type D Wild-Type Strain CN1020 and Its Genetically Modified Derivatives.$h[electronic resource]
260 $c2015
520 $aAbstract Clostridium perfringens type D causes enterotoxemia in sheep and goats. The disease is mediated by epsilon toxin (ETX), which affects the cerebrovascular endothelium, increasing vascular permeability and leading to cerebral edema. In the present study, we compared the distribution and severity of the cerebrovascular changes induced in lambs by C. perfringens type D strain CN1020, its isogenic etx null mutant, and the ETX-producing complemented mutant. We also applied histochemical and immunohistochemical markers to further characterize the brain lesions induced by ETX. Both ETX-producing strains induced extensive cerebrovascular damage that did not differ significantly between each other in nature, neuroanatomic distribution, or severity. By contrast, lambs inoculated with the etx mutant or sterile, nontoxic culture medium did not develop detectable brain lesions, confirming that the neuropathologic effects observed in these infections are dependent on ETX production. Lambs treated with the wild-type and complemented strains showed perivascular and mural vascular edema, as well as serum albumin extravasation, particularly severe in the cerebral white matter, midbrain, medulla oblongata, and cerebellum. Brains of animals inoculated with the ETX-producing strains showed decreased expression of glial fibrillary acidic protein and increased expression of aquaporin-4 in the end-feet processes of the astrocytes around blood vessels. Early axonal injury was demonstrated with anti?amyloid precursor protein immunohistochemistry. Perivascular accumulation of macrophages/microglia with intracytoplasmic albumin globules was also observed in these animals. This study demonstrates that ETX is responsible for the major cerebrovascular changes in C. perfringens type D?induced disease.
650 $aENFERMEDADES DE LOS ANIMALES
700 1 $aGIANNITTI, F.
700 1 $aFINNIE, J.W.
700 1 $aMANAVIS, J. .
700 1 $aBEINGESSER, J.
700 1 $aADAMS ,V.
700 1 $aROOD, J.I.
700 1 $aUZAL, F.A.
773 $tVeterinary Pathology, 2015$gv.52.n.6. p.1250-1253, 2015.
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