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| Registros recuperados : 8 | |
| 1. |  | GERMÁN, S.; CAMPOS, P.; CHAVES, M.; MADARIAGA, R.; CERETTA, S.; HUERTA-ESPINO, J.; HERRERA-FOESSEL, S.; SINGH, R.P. Differential expression of partial resistance to wheat leaf rust in Mexico and the Southern Cone of America. In: INTERNATIONAL WHEAT CONFERENCE, 8., 2010, St. Petersburg, RU. Abstracts...: wheat genetics and breeding for biotic stresses; poster presentations. St. Petersburg: VIR, 2010. p. 285. Presentado originalmente en: INTERNATIONAL WHEAT CONFERENCE, 8., 2010, St. Petersburg, RU. Abstracts...: wheat genetics and breeding for biotic stresses; poster presentations. St. Petersburg: VIR, 2010. p. 285.| Biblioteca(s): INIA La Estanzuela. |
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| 2. | | HUERTA-ESPINO, J..; SINGH, R.P.; GERMAN, S.; McCALLUM , B.D.; PARK , R.F.; CHEN, W.Q.; BHARDWAJ, S.C.; GOYEAU, H. Global status of wheat leaf rust caused by Puccinia triticina. Euphytica, v.179, n. 1, p. 143-160, 2011.| Biblioteca(s): INIA La Estanzuela. |
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| 3. | | GUTIÉRREZ, L.; CAPETTINI, F.; FROS, D.; GERMÁN, S.; HERRERA, S.; HUERTA-ESPINO, J.; PEREYRA, S.; PEREZ, S.; SANDOVAL-ISLAS, S.; SINGH, R.; CASTRO, A. Identifying disease resistance QTLs in barley germplasm from Latin America. In: INTERNATIONAL PLANT AND GENOME CONFERENCE, 19., 2011, San Diego, CA. [Abstracts]. [s.l.: INTLPAG], 2011.| Biblioteca(s): INIA La Estanzuela. |
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| 4. | | CALVO-SALAZAR, V.; SINGH, R. P.; HUERTA-ESPINO, J.; CRUZ-IZ QUIERDO, S.; LOBATO-ORTIZ, R.; SANDOVAL-ISLAS, S.; VARGAS-HERNÁNDEZ, M.; GERMAN, S.; SILVA, P.; BASNET, B. R.; LAN, C. X.; HERRERA-FOESSEL, S. A. Genetic Analysis of Resistance to Leaf Rust and Yellow Rust in Spring Wheat Cultivar Kenya Kongoni. Plant Disease, v. 99, no.1153-1160, 2015. Article history: Accepted for publication 9 February 2015/Published Online:23 Jun 2015.| Biblioteca(s): INIA La Estanzuela. |
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| 5. | | GUTIÉRREZ, L.; BERBERIAN, N.; CAPETTINI, F.; FALCIONI, E.; FROS, D.; GERMAN, S.; HAYES, P. M.; HUERTA-ESPINO, J.; HERRERA, S.; PEREYRA, S.; PEREZ, C.; SANDOVAL-ISLAS, S.; SINGH, R.; CASTRO, A. Genome-wide association mapping identifies disease resistance QTLs in barley germplasm from Latin America: chapter 18. In: INTERNATIONAL BARLEY GENETICS SYMPOSIUM, 11., 2012, Hangzhou, CN. Advance in barley sciences: proceedings. Dordrecht: Zhejiang University Press/Springer, 2013. p. 209-215| Biblioteca(s): INIA La Estanzuela. |
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| 6. | | GUTIÉRREZ, L.; BERBERIAN, N.; CAPETTINI. F.; FALCIONI, E.; FROS, D.; GERMAN, S.; HAYES, P.; HUERTA-ESPINO, J.; HERRERA, S.; PEREYRA, S.; PEREZ, C.; SANDOVAL-ISLAS, S.; SINGH, R.; CASTRO, A. Identifying disease resistance QTLs in barley germplasm from Latin America using genome-wide association mapping. In: NORTH AMERICAN BARLEY RESEARCHERS WORKSHOP, 20., 2011, Corvallis, OR. Abstracts. Corvallis: OSU, 2011.| Biblioteca(s): INIA La Estanzuela. |
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| 7. | | GUTIÉRREZ, L.; GERMAN, S.; PEREYRA, S.; HAYES, P.M.; PÉREZ, C.A.; CAPETTINI, F.; LOCATELLI, A.; BERBERIAN, N.M.; FALCONI, E.E.; ESTRADA, R.; DARIO FROS; GONZA, V.; ALTAMIRANO, H.; HUERTA-ESPINO, J.; NEYRA, E.; ORJEDA, G.; SANDOVAL-ISLAS, S.; SINGH, R.; TURKINGTON, K.; CASTRO, A.J. Multi-environment multi-QTL association mapping identifies disease resistance QTL in barley germplasm from Latin America. Theoretical Applied Genetics, 2015, v.128, n.3 p.501-516. Article history: Received 15 October 2014 / Accepted 17 December 2014 / First Online 30 December 2014.| Biblioteca(s): INIA La Estanzuela. |
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| 8. | | LEWIS ,C.M.; PERSOONS, A.; BEBBER, D.P.; KIGATHI, R.N.; MAINTZ, J.; FINDLAY, K.; BUENO-SANCHO, V.; CORREDOR-MORENO, P.; HARRINGTON, S.A.; KANGARA, N.; BERLIN, A.; GARCIA, R.; GERMAN, S.; HANZALOVÁ, A.; HODSON, D.P.; HOVMØLLER, M.S.; HUERTA-ESPINO, J.; IMTIAZ, M.; MIRZA, J.I.; JUSTESEN, A.F.; NIKS, R.E.; OMRANI, A.; PATPOUR, M.; PRETORIUS ,Z.A.; ROOHPARVAR, R.; SELA, H.; SINGH, R.P.; STEFFENSON ,B.; VISSER, B.; FENWICK, P.M.; THOMAS, J.; WULFF, B.B.H.; SAUNDERS, D.G.O. Potential for re-emergence of wheat stem rust in the United Kingdom. Veterinary Pathology [Vet Pathol], 2018 Sep 24, p. 300985818798117.OPEN ACCESS. Article history: Date Created: 20181002 //Latest Revision: 20181003.| Biblioteca(s): INIA La Estanzuela. |
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| Registros recuperados : 8 | |
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 | Acceso al texto completo restringido a Biblioteca INIA La Estanzuela. Por información adicional contacte bib_le@inia.org.uy. |
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Registro completo
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Biblioteca (s) : |
INIA La Estanzuela. |
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Fecha actual : |
02/03/2017 |
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Actualizado : |
07/03/2017 |
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Autor : |
GARCIA, J.P; GIANNITTI, F.; FINNIE, J.W.; MANAVIS, J. .; BEINGESSER, J.; ADAMS ,V.; ROOD, J.I.; UZAL, F.A. |
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Afiliación : |
FEDERICO GIANNITTI, INIA (Instituto Nacional de Investigación Agropecuaria), Uruguay. |
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Título : |
Comparative Neuropathology of Ovine Enterotoxemia Produced by Clostridium perfringens Type D Wild-Type Strain CN1020 and Its Genetically Modified Derivatives. |
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Fecha de publicación : |
2015 |
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Fuente / Imprenta : |
Veterinary Pathology, 2015, v.52.n.6. p.1250-1253, 2015. |
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Idioma : |
Inglés |
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Contenido : |
Abstract
Clostridium perfringens type D causes enterotoxemia in sheep and goats. The disease is mediated by epsilon toxin (ETX), which affects the cerebrovascular endothelium, increasing vascular permeability and leading to cerebral edema. In the present study, we compared the distribution and severity of the cerebrovascular changes induced in lambs by C. perfringens type D strain CN1020, its isogenic etx null mutant, and the ETX-producing complemented mutant. We also applied histochemical and immunohistochemical markers to further characterize the brain lesions induced by ETX. Both ETX-producing strains induced extensive cerebrovascular damage that did not differ significantly between each other in nature, neuroanatomic distribution, or severity. By contrast, lambs inoculated with the etx mutant or sterile, nontoxic culture medium did not develop detectable brain lesions, confirming that the neuropathologic effects observed in these infections are dependent on ETX production. Lambs treated with the wild-type and complemented strains showed perivascular and mural vascular edema, as well as serum albumin extravasation, particularly severe in the cerebral white matter, midbrain, medulla oblongata, and cerebellum. Brains of animals inoculated with the ETX-producing strains showed decreased expression of glial fibrillary acidic protein and increased expression of aquaporin-4 in the end-feet processes of the astrocytes around blood vessels. Early axonal injury was demonstrated with anti?amyloid precursor protein immunohistochemistry. Perivascular accumulation of macrophages/microglia with intracytoplasmic albumin globules was also observed in these animals. This study demonstrates that ETX is responsible for the major cerebrovascular changes in C. perfringens type D?induced disease. MenosAbstract
Clostridium perfringens type D causes enterotoxemia in sheep and goats. The disease is mediated by epsilon toxin (ETX), which affects the cerebrovascular endothelium, increasing vascular permeability and leading to cerebral edema. In the present study, we compared the distribution and severity of the cerebrovascular changes induced in lambs by C. perfringens type D strain CN1020, its isogenic etx null mutant, and the ETX-producing complemented mutant. We also applied histochemical and immunohistochemical markers to further characterize the brain lesions induced by ETX. Both ETX-producing strains induced extensive cerebrovascular damage that did not differ significantly between each other in nature, neuroanatomic distribution, or severity. By contrast, lambs inoculated with the etx mutant or sterile, nontoxic culture medium did not develop detectable brain lesions, confirming that the neuropathologic effects observed in these infections are dependent on ETX production. Lambs treated with the wild-type and complemented strains showed perivascular and mural vascular edema, as well as serum albumin extravasation, particularly severe in the cerebral white matter, midbrain, medulla oblongata, and cerebellum. Brains of animals inoculated with the ETX-producing strains showed decreased expression of glial fibrillary acidic protein and increased expression of aquaporin-4 in the end-feet processes of the astrocytes around blood vessels. Early axonal injury was demonstrated wi... Presentar Todo |
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Thesagro : |
ENFERMEDADES DE LOS ANIMALES. |
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Asunto categoría : |
-- |
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Marc : |
LEADER 02512naa a2200217 a 4500
001 1056750
005 2017-03-07
008 2015 bl uuuu u00u1 u #d
100 1 $aGARCIA, J.P
245 $aComparative Neuropathology of Ovine Enterotoxemia Produced by Clostridium perfringens Type D Wild-Type Strain CN1020 and Its Genetically Modified Derivatives.$h[electronic resource]
260 $c2015
520 $aAbstract Clostridium perfringens type D causes enterotoxemia in sheep and goats. The disease is mediated by epsilon toxin (ETX), which affects the cerebrovascular endothelium, increasing vascular permeability and leading to cerebral edema. In the present study, we compared the distribution and severity of the cerebrovascular changes induced in lambs by C. perfringens type D strain CN1020, its isogenic etx null mutant, and the ETX-producing complemented mutant. We also applied histochemical and immunohistochemical markers to further characterize the brain lesions induced by ETX. Both ETX-producing strains induced extensive cerebrovascular damage that did not differ significantly between each other in nature, neuroanatomic distribution, or severity. By contrast, lambs inoculated with the etx mutant or sterile, nontoxic culture medium did not develop detectable brain lesions, confirming that the neuropathologic effects observed in these infections are dependent on ETX production. Lambs treated with the wild-type and complemented strains showed perivascular and mural vascular edema, as well as serum albumin extravasation, particularly severe in the cerebral white matter, midbrain, medulla oblongata, and cerebellum. Brains of animals inoculated with the ETX-producing strains showed decreased expression of glial fibrillary acidic protein and increased expression of aquaporin-4 in the end-feet processes of the astrocytes around blood vessels. Early axonal injury was demonstrated with anti?amyloid precursor protein immunohistochemistry. Perivascular accumulation of macrophages/microglia with intracytoplasmic albumin globules was also observed in these animals. This study demonstrates that ETX is responsible for the major cerebrovascular changes in C. perfringens type D?induced disease.
650 $aENFERMEDADES DE LOS ANIMALES
700 1 $aGIANNITTI, F.
700 1 $aFINNIE, J.W.
700 1 $aMANAVIS, J. .
700 1 $aBEINGESSER, J.
700 1 $aADAMS ,V.
700 1 $aROOD, J.I.
700 1 $aUZAL, F.A.
773 $tVeterinary Pathology, 2015$gv.52.n.6. p.1250-1253, 2015.
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